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Displaying 20 of 109 results for "OSM"
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  1. Oncostatin M (OSM) and Leukemia Inhibitory factor (LIF) treatment of osteoblasts via LIF and OSM receptors BioProject

    ID: PRJNA325836

    Keywords: Transcriptome or Gene expression

    Access Type: download

  2. Gene expression of HCT116 cells after stimulation with oncostatin M (OSM) BioProject

    ID: PRJNA231619

    Keywords: Transcriptome or Gene expression

    Access Type: download

  3. Gene-expression analysis of Oncostatin-M (OSM) signalling in cervical squamous cell carcinomas over-expressing the Oncostatin-M receptor (OSMR) BioProject

    ID: PRJNA138587

    Keywords: Transcriptome or Gene expression

    Access Type: download

  4. ng by arrayof HCT116 cells after stimulation with oncostatin M (OSM) against untreated controls to study the role of OSM in intestinal inflammati... ArrayExpress

    ID: E-GEOD-53295

    Description: ofiles in intestinal epithelial cells (IEC) after OSM stimulation, we have employed whole genome microarray expression profiling as a discovery platform to identify relevant genes which are potentially of intere...

  5. Oncostatin M effects in IMR90 cells BioProject

    ID: PRJNA141247

    Keywords: Transcriptome or Gene expression

    Access Type: download

  6. Oncostatin M effects in IMR90 cells ArrayExpress

    ID: E-GEOD-29655

    Description: re we show that JAK2 V617F promotes expression of oncostatin M (OSM) in neoplastic myeloid cells. Correspondingly, OSM was found to be overexpressed in the BM and elevated in the serum of patients with JAK2 V617F+ MPN. In addition, OSM secreted by JAK2 V617F+ cells stimulated growth of fibroblasts and microvascular endothelial cells and induced the production of angiogenic and profibrogenic cytokines (HGF, VEGF, and SDF-1) in BM fibroblasts. All effects of MPN cell-derived OSM were blocked by a neutralizi...

  7. Gene-expression analysis of Oncostatin-M (OSM) signalling in cervical squamous cell carcinomas over-expressing the Oncostatin-M receptor (OSMR) ArrayExpress

    ID: E-GEOD-27480

    Description: The type II Oncostatin M receptor (OSMR) serves as the main binding site for the pleiotropic cytokine OSM...

  8. The genes regulated by JAK2 and Lnc-BM in breast cancer brain metastasis cells (231-BM, derived from MDA-MB-231 cells) treated with Oncostatin M (OSM) BioProject

    ID: PRJNA316112

    Keywords: Transcriptome or Gene expression

    Access Type: download

  9. Gene-expression analysis of Oncostatin-M (OSM) signalling in cervical squamous cell carcinomas over-expressing the Oncostatin-M receptor (OSMR) OmicsDI

    ID: E-GEOD-27480

    Date Released: 10-12-2011

    Description: The type II Oncostatin M receptor (OSMR) serves as the main binding site for the pleiotropic cytokine OSM...

  10. Oncostatin M Regulation of Gene Expression in Breast Tumour Cells. BioProject

    ID: PRJNA96803

    Keywords: Transcriptome or Gene expression

    Access Type: download

  11. Oncostatin M effects in IMR90 cells OmicsDI

    ID: E-GEOD-29655

    Date Released: 02-11-2013

    Description: re we show that JAK2 V617F promotes expression of oncostatin M (OSM) in neoplastic myeloid cells. Correspondingly, OSM was found to be overexpressed in the BM and elevated in the serum of patients with JAK2 V617F+ MPN. In addition, OSM secreted by JAK2 V617F+ cells stimulated growth of fibroblasts and microvascular endothelial cells and induced the production of angiogenic and profibrogenic cytokines (HGF, VEGF, and SDF-1) in BM fibroblasts. All effects of MPN cell-derived OSM were blocked by a neutralizi...

  12. ng by arrayof HCT116 cells after stimulation with oncostatin M (OSM) against untreated controls to study the role of OSM in intestinal inflammati... OmicsDI

    ID: E-GEOD-53295

    Date Released: 04-30-2015

    Description: ofiles in intestinal epithelial cells (IEC) after OSM stimulation, we have employed whole genome microarray expression profiling as a discovery platform to identify relevant genes which are potentially of intere...

  13. Oncostatin M promotes cancer cell plasticity through cooperative STAT3-SMAD3 signaling BioProject

    ID: PRJNA357114

    Keywords: Transcriptome or Gene expression

    Access Type: download

  14. Transcriptional profiling of HEPG2 cells after treatment with TRIB1-inducing compounds BioProject

    ID: PRJNA248003

    Keywords: Transcriptome or Gene expression

    Access Type: download

    dataset.description: of HEPG2 cells with TRIB1-inducing compounds and Oncostatin M Transcriptional response of HEPG2 cells was measured after treatment with TRIB1-inducing compound BRD8518 an...
  15. Transcription profiling by array of neonatal rat cardiomyocytes stimulated by oncostatin M against untreated controls ArrayExpress

    ID: E-MTAB-2560

    Description: nscriptional changes in cardiomyocytes induced by Oncostatin M treatment. The rationale of this experiment is described in [Kubin, T., et al. Oncostatin M

  16. Microarray analysis of Huh7 cells treated with IFNa2, OSM or IFNa2 combined with OSM BioProject

    ID: PRJNA109957

    Keywords: Transcriptome or Gene expression

    Access Type: download

  17. Transcription profiling time series of human epidermal keratinocytes treated with Oncostatin-M ArrayExpress

    ID: E-GEOD-2822

    Description: Cultured epidermal keratinocytes treated with OsM 1, 4, 24 & 48hrs, and Skinethic epidermal substitutes treated 1, 4, 24, 48h & 7days, each with untreated control Experiment Ove...

  18. ONCM_BOVIN UniProt:Swiss-Prot

    ID: P53346

    Description: Oncostatin-M N-linked (GlcNAc...)

  19. Microarray analysis of Huh7 cells treated with IFNa2, OSM or IFNa2 combined with OSM ArrayExpress

    ID: E-GEOD-13046

    Description: OSM increases the antiviral effect of IFNα in Huh7 cells infected with hepatitis A virus (HAV) or HCV replicon and synergizes with...

  20. Leukemia Inhibitory Factor in C26 Cancer Cachexia ArrayExpress

    ID: E-GEOD-68827

    Description: or AP-1. Of the gp130 family members IL-11, IL-6, oncostatin M (OSM), and leukemia inhibitory factor (LIF), only OSM and LIF were sufficient to activate the STAT reporter in myotubes. A LIF blocking antibody abolished C26 CM-induced STAT reporter activation STAT3 phosphorylation and myotube atrophy, but blocking antibodies to IL-6 or OSM did not. JAK2 inhibitors also blocked the C26 CM-induced STAT reporter activation, STAT3 phosphorylation, and atrophy in myotubes. LIF at levels found in the C26 CM was sufficient for STAT reporter activation and ...


Displaying 20 of 109 results for "OSM"