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Displaying 9 of 9 results for "NTSR2"
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  1. NTR2_RAT UniProt:Swiss-Prot

    ID: Q63384

    Description: Neurotensin receptor type 2 Extracellular Helical; Name=1 Cytoplasmic Helical; Name=

  2. The crystal structure of the neurotensin receptor NTS1 in complex with neurotensin (8-13) PDB

    ID: PDB:4GRV

    Description: Neurotensin receptor type 1, Neurotensin 8-13

  3. NTR1_RAT UniProt:Swiss-Prot

    ID: P20789

    Description: Neurotensin receptor type 1 Extracellular Helical; Name=1 Cytoplasmic Helical; Name=2 Extrace...

  4. Structure of active-like neurotensin receptor PDB

    ID: PDB:4XEE

    Description: Neurotensin receptor type 1, lysozyme chimera (E.C.3.2.1.17), Neurotensin (8-13)

  5. NTR1_MOUSE UniProt:Swiss-Prot

    ID: O88319

    Description: Neurotensin receptor type 1 Extracellular Helical; Name=1 Cytoplasmic Helical; Name=2 Extrace...

  6. Structure of active-like neurotensin receptor PDB

    ID: PDB:4XES

    Description: neurotensin receptor subtype 1, lysozyme chimera (E.C.3.2.1.17), Neurotensin (8-13)

  7. Pain microarray analysis 2 BioProject

    ID: PRJNA98047

    Keywords: Transcriptome or Gene expression

    Access Type: download

  8. Pain microarray analysis 1 BioProject

    ID: PRJNA98261

    Keywords: Transcriptome or Gene expression

    Access Type: download

    dataset.description: GABA, cholecystokinin, histamine, serotonin, and neurotensin systems) in the LDH ipsilateral to the neonatally-injured paw. The largest aberration in gene expression, however, was observed during inflammation of the neonatally injured hindpaws in the ipsilateral LDH, which included thirty-six genes (encoding numerous members of glutamate, serotonin, GABA, calcitonin gene-related peptide, neurotrophin, and interleukin systems). These findings suggest that changes in gene expression may be involved in the long-term nociceptive effects of neonatal noxious insult at the spinal level. Keywords: gene expression analysis via microarray Overall design: To gain an insight into the long-term molecular changes involved in the above-described long-term nociceptive effects of neonatal noxious insult at the spinal level, we performed DNA microarray analysis (using microarrays containing oligo-probes for 205 genes encoding receptors and transporters for glutamate, GABA, and amine neurotransmitters, precursors and receptors for neuropeptides, and neurotrophins, cytokines and their receptors) to compare gene expression profiles in the lumbar spinal dorsal horn (LDH) of ...
  9. Pain microarray analysis 2 GEMMA

    ID: 1638

    Keywords: functional genomics

    Description: GABA, cholecystokinin, histamine, serotonin, and neurotensin systems) in the LDH ipsilateral to the neonatally-injured paw. The largest aberration in gene expression, however, was observed during inflammation of the neonatally injured hindpaws in the ipsilateral LDH, which included thirty-six genes (encoding numerous members of glutamate, serotonin, GABA, calcitonin gene-related peptide, neurotrophin, and interleukin systems). These findings suggest that changes in gene expression may be involved in the long-term nociceptive effects of neonatal noxious insult at the spinal level. Last Updated (by provider): Mar 20 2007 Contributers: Michael S Lidow Fang He Jie Bai Svetlana I Novikova...


Displaying 9 of 9 results for "NTSR2"