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Displaying 7 of 7 results for "LMCD1"
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  1. LMCD1_BOVIN UniProt:Swiss-Prot

    ID: Q17QE2

    Description: LIM and cysteine-rich domains protein 1 PET LIM zinc-binding 1 LIM zinc-binding 2 Cys-rich Phosphoserine

    gene.name: LMCD1
  2. LMCD1_MOUSE UniProt:Swiss-Prot

    ID: Q8VEE1

    Description: LIM and cysteine-rich domains protein 1 PET LIM zinc-binding 1 LIM zinc-binding 2 Cys-rich Phosphoserine

    gene.name: Lmcd1
  3. LMCD1_PIG UniProt:Swiss-Prot

    ID: Q5PXT2

    Description: LIM and cysteine-rich domains protein 1 PET LIM zinc-binding 1 LIM zinc-binding 2 Cys-rich Phosphoserine

    gene.name: LMCD1
  4. Expression Profiling of Spalax ehrenbergi: bioprospecting for hypoxia tolerance BioProject

    ID: PRJNA93939

    Keywords: Transcriptome or Gene expression

    Access Type: download

    dataset.description: factor 3 (Atf3), LIM and cysteine-rich domains 1 (Lmcd1) and syndecan 2 (Sdc2). These results support the hypothesis that Spalax is variably adapted to fluctuating oxygen tension. Differences may involve very specific metabolic pathways and functional adaptations at the structural and molecular levels. Elucidation of the natural variation and evolutionary changes under hypoxia within this superspecies may have biomedical applications in ischemic syndromes and cancer. Keywords: hypoxia, Spalax, cDNA microarray Overall design: 12 animalsin total, 2 species (s52;S. galili, s60; S. judaei), 2 conditions (normoxia, hypoxia), 3 biological replicates. Loop design with dye swap....
  5. Gene expression profile of human heart failure from different etiologies ArrayExpress

    ID: E-GEOD-2656

    Description: iling hearts. The LIM and cysteine rich domain 1 (LMCD1) gene, in particular, was significantly down-regulated in all HF samples. This novel finding suggests that the LMCD 1 is a universal biomarker for end-stage HF. Other LIM domain genes were also down-regulated but only in non-familial dilated forms of cardiomyopathy. This is probably due to the fact that down-regulation of LIM protein expression may disrupt the cytoskeletal architecture, leading to dilated cardiomyopathy. In addition to identifying the LIM domain genes as possible regulatory genes involved in HF, we also demonstrated that the gene expression profile was able to classify multiple HF patients groups. This paper is the first to examine viral-induced cardiomyopathy, doxorubicin toxicity cardiomyopathy and perimartum cardiomyopathy and to perform the clustering analysis of those HF types providing new insights into human HF....

  6. Gene expression profile of human heart failure from different etiologies BioProject

    ID: PRJNA92123

    Keywords: Transcriptome or Gene expression

    Access Type: download

    dataset.description: iling hearts. The LIM and cysteine rich domain 1 (LMCD1) gene, in particular, was significantly down-regulated in all HF samples. This novel finding suggests that the LMCD 1 is a universal biomarker for end-stage HF. Other LIM domain genes were also down-regulated but only in non-familial dilated forms of cardiomyopathy. This is probably due to the fact that down-regulation of LIM protein expression may disrupt the cytoskeletal architecture, leading to dilated cardiomyopathy. In addition to identifying the LIM domain genes as possible regulatory genes involved in HF, we also demonstrated that the gene expression profile was able to classify multiple HF patients groups. This paper is the first to examine viral-induced cardiomyopathy, doxorubicin toxicity cardiomyopathy and perimartum cardiomyopathy and to perform the clustering analysis of those HF types providing new insights into human HF. Keywords: other...
  7. Gene expression profile of human heart failure from different etiologies OmicsDI

    ID: E-GEOD-2656

    Date Released: 10-18-2011

    Description: iling hearts. The LIM and cysteine rich domain 1 (LMCD1) gene, in particular, was significantly down-regulated in all HF samples. This novel finding suggests that the LMCD 1 is a universal biomarker for end-stage HF. Other LIM domain genes were also down-regulated but only in non-familial dilated forms of cardiomyopathy. This is probably due to the fact that down-regulation of LIM protein expression may disrupt the cytoskeletal architecture, leading to dilated cardiomyopathy. In addition to identifying the LIM domain genes as possible regulatory genes involved in HF, we also demonstrated that the gene expression profile was able to classify multiple HF patients groups. This paper is the first to examine viral-induced cardiomyopathy, doxorubicin toxicity cardiomyopathy and perimartum cardiomyopathy and to perform the clustering analysis of those HF types providing new insights into human HF....


Displaying 7 of 7 results for "LMCD1"