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Displaying 10 of 10 results for "FANCF"
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  1. Crystal structure of Human FancF Protein that Functions in the Assembly of a DNA Damage Signaling Complex PDB

    ID: PDB:2IQC

    Description: Fanconi anemia group F protein

  2. Crystal structure of the C-terminal segment of FANCM in complex with FAAP24 PDB

    ID: PDB:4M6W

    Description: Fanconi anemia group M protein (E.C.3.6.4.13), Fanconi ...

  3. FANCF_MOUSE UniProt:Swiss-Prot

    ID: E9Q5Z5

    Description: Fanconi anemia group F protein

  4. Solution Structure of (HhH)2 domain of human FAAP24 PDB

    ID: PDB:2M9N

    Description: Fanconi anemia-associated protein of 24 kDa

  5. Solution Structure of ERCC4 domain of human FAAP24 PDB

    ID: PDB:2M9M

    Description: Fanconi anemia-associated protein of 24 kDa

  6. Oxymetholone therapy of Fanconi anemia suppresses osteopontin transcription and induces hematopoietic stem cell cycling ArrayExpress

    ID: E-GEOD-58724

    Description: ytometry analysis, surprisingly we noticed that a group of genes involved in immune responses and inflammation, comprising Cfp (Properdin), Socs2, Ccr1, Ccr2, Ccr5, Chga (Chromogranin A), Ifi30 (Interferon Gamma-Inducible Protein 30), Lgmn, Txn, and Sell (selectin L), were up-regulated in Fancd2−/− KSL cells. We therefore hypothesize that some genes up-regulated in FA HSPCs may be part of an innate immune response to DNA damage. In addition, whole bone marrow cells were also analyzed in parallel with KSL cells. As compared to whole bone marrow cells, the genes enriched in KSL cells in wild-type mice were listed in details in the corresponding publication. This information can be a good resource for the future gene expression analysis of HSPCs. Finally, we compared the gene expression profiles of early progenitors between OXM-treated and placebo-treated mice. There were no significant differences at all in gene expression between OXM-treated wild-type erythroid progenitors and their placebo-treated wild-type counterparts, with no genes displaying an expression change higher than 1.2 fold. Importantly, no up-regulation of EPO-inducible genes such as Socs1, Socs2, Socs3, and Cish was seen in wild-type mice treated with OXM. Furthermore, there was no differential expression of the well-known EPO target transferrin receptor or any other major players of the Epo-R signaling network such as Bcl2l1, Cdc25a,...

  7. FANCG_HUMAN UniProt:Swiss-Prot

    ID: O15287

    Description: Fanconi anemia group G protein TPR 1 TPR 2 TPR 3 TPR 4 Phosphoserine In FA...

  8. Oxymetholone therapy of Fanconi anemia suppresses osteopontin transcription and induces hematopoietic stem cell cycling BioProject

    ID: PRJNA253277

    Keywords: Transcriptome or Gene expression

    Access Type: download

  9. Integrative Molecular Profiling in Leiomyosarcoma ArrayExpress

    ID: E-GEOD-17555

    Description: formation not available.) Sex/Mating type: M(ale)/F(emale) Experiment type: aCGH or Expression type II A disease state experiment design type is where the state of some disease such as infection, pathology, syndrome, etc is studied....

  10. Integrative Molecular Profiling in Leiomyosarcoma BioProject

    ID: PRJNA118653

    Keywords: Other

    Access Type: download

    dataset.description: formation not available.) Sex/Mating type: M(ale)/F(emale) Experiment type: aCGH or Expression type II Overall design: A disease state experiment design type is where the state of some disease such as infection, pathology, syndrome, etc is studied....

Displaying 10 of 10 results for "FANCF"