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Displaying 10 of 10 results for "COL17A1"
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  1. Hair follicle aging is driven by transepidermal stem cell elimination via COL17A1 proteolysis BioProject

    ID: PRJNA295214

    Keywords: Transcriptome or Gene expression

    Access Type: download

  2. COHA1_CANLF UniProt:Swiss-Prot

    ID: Q9N281

    Description: Collagen alpha-1(XVII) chain 120 kDa linear IgA disease antigen homolog Cy...

  3. COHA1_BOVIN UniProt:Swiss-Prot

    ID: A6QPB3

    Description: Collagen alpha-1(XVII) chain 120 kDa linear IgA disease antigen homolog Cy...

  4. COHA1_CHICK UniProt:Swiss-Prot

    ID: Q90584

    Description: Collagen alpha-1(XVII) chain 120 kDa linear IgA disease antigen homolog Cy...

  5. COHA1_MESAU UniProt:Swiss-Prot

    ID: Q9JMH4

    Description: Collagen alpha-1(XVII) chain 120 kDa linear IgA disease antigen homolog Cy...

  6. Additional file 2: of In epithelial cancers, aberrant COL17A1 promoter methylation predicts its misexpression and increased invasion Figshare

    ID: doi:10.6084/M9.FIGSHARE.C.3646622_D1

    Release Date: 12-16-2016

    Description: COL17A1 promoter methylation levels (β-values, TCGA Illumina Infinium Human Methylation 450). (XLSX 231 kb)

  7. Transcription profiling of mouse back skin fromK14-DN-Clim OmicsDI

    ID: E-GEOD-8227

    Date Released: 05-02-2014

    Description: causes decreased expression of adhesion molecule BP180 and defective hemidesmosomes, leading to detachment of corneal epithelium from the underlying stroma, which in turn causes blisters, wounds and an inflammatory response. After a period of epithelial thinning, the corneal epithelium undergoes differentiation to an epidermis-like structure. The K14-DN-Clim mice also develop progressive hair loss due to dysfunctional hair follicles that fail to generate hair shafts. The number of hair follicle stem cells is decreased by at least 50% in K14-DN-Clim mice, indicating that Clims are required for hair follicle stem cell maintenance. We hypothesize that Clim2 is an essential co-factor for the LIM homeodomain factor Lhx2, which was previously shown to play a role in hair follicle stem cell mainten...

  8. Transcription profiling of mouse back skin fromK14-DN-Clim ArrayExpress

    ID: E-GEOD-8227

    Description: causes decreased expression of adhesion molecule BP180 and defective hemidesmosomes, leading to detachment of corneal epithelium from the underlying stroma, which in turn causes blisters, wounds and an inflammatory response. After a period of epithelial thinning, the corneal epithelium undergoes differentiation to an epidermis-like structure. The K14-DN-Clim mice also develop progressive hair loss due to dysfunctional hair follicles that fail to generate hair shafts. The number of hair follicle stem cells is decreased by at least 50% in K14-DN-Clim mice, indicating that Clims are required for hair follicle stem cell maintenance. We hypothesize that Clim2 is an essential co-factor for the LIM homeodomain factor Lhx2, which was previously shown to play a role in hair follicle stem cell mainten...

  9. Expression profiling of K14-DN-Clim mouse backskin BioProject

    ID: PRJNA101153

    Keywords: Transcriptome or Gene expression

    Access Type: download

    dataset.description: causes decreased expression of adhesion molecule BP180 and defective hemidesmosomes, leading to detachment of corneal epithelium from the underlying stroma, which in turn causes blisters, wounds and an inflammatory response. After a period of epithelial thinning, the corneal epithelium undergoes differentiation to an epidermis-like structure. The K14-DN-Clim mice also develop progressive hair loss due to dysfunctional hair follicles that fail to generate hair shafts. The number of hair follicle stem cells is decreased by at least 50% in K14-DN-Clim mice, indicating that Clims are required for hair follicle stem cell maintenance. We hypothesize that Clim2 is an essential co-factor for the LIM homeodomain factor Lhx2, which was previously shown to play a role in hair follicle stem cell mainten...
  10. Expression profiling of K14-DN-Clim mouse backskin GEMMA

    ID: 567

    Keywords: functional genomics

    Description: causes decreased expression of adhesion molecule BP180 and defective hemidesmosomes, leading to detachment of corneal epithelium from the underlying stroma, which in turn causes blisters, wounds and an inflammatory response. After a period of epithelial thinning, the corneal epithelium undergoes differentiation to an epidermis-like structure. The K14-DN-Clim mice also develop progressive hair loss due to dysfunctional hair follicles that fail to generate hair shafts. The number of hair follicle stem cells is decreased by at least 50% in K14-DN-Clim mice, indicating that Clims are required for hair follicle stem cell maintenance. We hypothesize that Clim2 is an essential co-factor for the LIM homeodomain factor Lhx2, which was previously shown to play a role in hair follicle stem cell mainten...


Displaying 10 of 10 results for "COL17A1"