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Displaying 20 of 32 results for "CD22"
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  1. Clostridioides difficile CD22 : Clostridioides difficile CD22 Genome sequencing and assembly BioProject

    ID: PRJNA85691

    Keywords: Genome sequencing and assembly

    Access Type: download

  2. HGAL Enhances BCR-mediated Syk Activation, Leading to Lymphoid Hyperplasia and Amyloidosis ArrayExpress

    ID: E-GEOD-35400

    Description: Gene expression data from CD22+B220+ FACS-purified splenocytes of adult Sca1-HGAL knock-in CBAxC57BL/6J mice or wild-type littermates. Transgenic mice expres...

  3. To understand how glycosylation on human B cells is regulated during differentiation and activation ArrayExpress

    ID: E-GEOD-31932

    Description: ne of well-known examples is B cell regulation by CD22 through its association with sialylated glycans. While recent studies uncovered that B cell activation leads to loss of high affinity glycan ligands for human CD22 (Neu5Ac-a2,6-Gal-B1,4-GlcNAc(6-sulfo)), the gene expression profile of human B cells before and after activation is unknown. Therefore we would like to perform gene expressio...

  4. Cancer arises through reprogramming-like processes in mice BioProject

    ID: PRJNA153449

    Keywords: Transcriptome or Gene expression

    Access Type: download

    dataset.description: Gene expression data from CD22+B220+ FACS-purified splenocytes of adult wild-type, Sca1-Bcl6(floxed) and Sca1-Bcl6(Δ) knock-in CBAxC57BL/6J mice. Overall de...
  5. HGAL Enhances BCR-mediated Syk Activation, Leading to Lymphoid Hyperplasia and Amyloidosis BioProject

    ID: PRJNA152787

    Keywords: Transcriptome or Gene expression

    Access Type: download

    dataset.description: Gene expression data from CD22+B220+ FACS-purified splenocytes of adult Sca1-HGAL knock-in CBAxC57BL/6J mice or wild-type littermates. Overall design: Transg...
  6. To understand how glycosylation on human B cells is regulated during differentiation and activation OmicsDI

    ID: E-GEOD-31932

    Date Released: 09-14-2011

    Description: ne of well-known examples is B cell regulation by CD22 through its association with sialylated glycans. While recent studies uncovered that B cell activation leads to loss of high affinity glycan ligands for human CD22 (Neu5Ac-a2,6-Gal-B1,4-GlcNAc(6-sulfo)), the gene expression profile of human B cells before and after activation is unknown. Therefore we would like to perform gene expressio...

  7. Cancer arises through reprogramming-like processes in mice ArrayExpress

    ID: E-GEOD-36503

    Description: Gene expression data from CD22+B220+ FACS-purified splenocytes of adult wild-type, Sca1-Bcl6(floxed) and Sca1-Bcl6(Δ) knock-in CBAxC57BL/6J mice. The Bcl6 f...

  8. CD22_GORGO UniProt:Swiss-Prot

    ID: Q9N1E4

    Description: B-cell receptor CD22 Extracellular Ig-like V-type Ig-like C2-type 1 Ig-like C2-type 2 Sialic acid N-linked (GlcNAc...) N-linked (GlcNAc...) N-linke...

  9. The cellular origin and malignant transformation of Waldenström's Macroglobulinemia [gene expression] ArrayExpress

    ID: E-GEOD-61597

    Description: clone was highly deregulated as compared to CD25-CD22+ normal B-cells, significantly less genes were differentially expressed and specific WM pathways down-regulated while comparing the transcriptome of the Waldenström’s clone vs. its normal phenotypic counterpart: CD25+CD22+dim B-cells. The frequency of specific copy number abnormalities [+4, del(6q23.3-6q25.3), +12, and +18q11-18q23] progressively increased from IgM MGUS and smoldering WM vs. symptomatic WM (18% vs. 20% and 73%, respectively; P =.008), suggesting a multistep transformation of clonal B-cells that albeit benign (i.e.: IgM MGUS and smoldering WM), already harbor the phenotypic and molecular signatures of the malignant Waldenström’s clone. Normal bone marrow CD25+ B-cells, Clonal B-Cells from IgM Monoclonal Gammopathy of Undetermined Significance, and Clonal B-Cells from Waldenström's Macroglobulinemia...

  10. The cellular origin and malignant transformation of Waldenström's Macroglobulinemia [copy number variation] ArrayExpress

    ID: E-GEOD-61650

    Description: clone was highly deregulated as compared to CD25-CD22+ normal B-cells, significantly less genes were differentially expressed and specific WM pathways down-regulated while comparing the transcriptome of the Waldenström’s clone vs. its normal phenotypic counterpart: CD25+CD22+dim B-cells. The frequency of specific copy number abnormalities [+4, del(6q23.3-6q25.3), +12, and +18q11-18q23] progressively increased from IgM MGUS and smoldering WM vs. symptomatic WM (18% vs. 20% and 73%, respectively; P =.008), suggesting a multistep transformation of clonal B-cells that albeit benign (i.e.: IgM MGUS and smoldering WM), already harbor the phenotypic and molecular signatures of the malignant Waldenström’s clone. Copy number analysis using the Affymetrix CytoScan 750K Array to study clonal B-cells from newly-diagnosed patients with IgM MGUS, smoldering, and symptomatic WM. Peripheral T-cells from paired and unpaired controls were also assessed....

  11. To understand how glycosylation on human B cells is regulated during differentiation and activation BioProject

    ID: PRJNA145029

    Keywords: Transcriptome or Gene expression

    Access Type: download

    dataset.description: ne of well-known examples is B cell regulation by CD22 through its association with sialylated glycans. While recent studies uncovered that B cell activation leads to loss of high affinity glycan ligands for human CD22 (Neu5Ac-a2,6-Gal-B1,4-GlcNAc(6-sulfo)), the gene expression profile of human B cells before and after activation is unknown. Therefore we would like to perform gene expressio...
  12. Cancer arises through reprogramming-like processes in mice OmicsDI

    ID: E-GEOD-36503

    Date Released: 03-14-2014

    Description: Gene expression data from CD22+B220+ FACS-purified splenocytes of adult wild-type, Sca1-Bcl6(floxed) and Sca1-Bcl6(Δ) knock-in CBAxC57BL/6J mice. The Bcl6 f...

  13. CD22_PANTR UniProt:Swiss-Prot

    ID: Q9N1E6

    Description: B-cell receptor CD22 Extracellular Ig-like V-type Ig-like C2-type 1 Ig-like C2-type 2 Sialic acid N-linked (GlcNAc...) N-linked (GlcNAc...) N-linke...

  14. CD22_PONPY UniProt:Swiss-Prot

    ID: Q9N1E3

    Description: B-cell receptor CD22 Extracellular Ig-like V-type Ig-like C2-type 1 Ig-like C2-type 2 Sialic acid N-linked (GlcNAc...) N-linked (GlcNAc...) N-linke...

  15. CD22_PANPA UniProt:Swiss-Prot

    ID: Q9N1E5

    Description: B-cell receptor CD22 Extracellular Ig-like V-type Ig-like C2-type 1 Ig-like C2-type 2 Sialic acid N-linked (GlcNAc...) N-linked (GlcNAc...) N-linke...

  16. HGAL Enhances BCR-mediated Syk Activation, Leading to Lymphoid Hyperplasia and Amyloidosis OmicsDI

    ID: E-GEOD-35400

    Date Released: 06-17-2013

    Description: Gene expression data from CD22+B220+ FACS-purified splenocytes of adult Sca1-HGAL knock-in CBAxC57BL/6J mice or wild-type littermates. Transgenic mice expres...

  17. The cellular origin and malignant transformation of Waldenström's Macroglobulinemia [copy number variation] BioProject

    ID: PRJNA261789

    Keywords: Variation

    Access Type: download

    dataset.description: clone was highly deregulated as compared to CD25-CD22+ normal B-cells, significantly less genes were differentially expressed and specific WM pathways down-regulated while comparing the transcriptome of the Waldenström’s clone vs. its normal phenotypic counterpart: CD25+CD22+dim B-cells. The frequency of specific copy number abnormalities [+4, del(6q23.3-6q25.3), +12, and +18q11-18q23] progressively increased from IgM MGUS and smoldering WM vs. symptomatic WM (18% vs. 20% and 73%, respectively; P =.008), suggesting a multistep transformation of clonal B-cells that albeit benign (i.e.: IgM MGUS and smoldering WM), already harbor the phenotypic and molecular signatures of the malignant Waldenström’s clone. Overall design: Copy number analysis using the Affymetrix CytoScan 750K Array to study clonal B-cells from newly-diagnosed patients with IgM MGUS, smoldering, and symptomatic WM. Peripheral T-cells from paired and unpaired controls were also assessed....
  18. The cellular origin and malignant transformation of Waldenström's Macroglobulinemia [gene expression] BioProject

    ID: PRJNA261632

    Keywords: Transcriptome or Gene expression

    Access Type: download

    dataset.description: clone was highly deregulated as compared to CD25-CD22+ normal B-cells, significantly less genes were differentially expressed and specific WM pathways down-regulated while comparing the transcriptome of the Waldenström’s clone vs. its normal phenotypic counterpart: CD25+CD22+dim B-cells. The frequency of specific copy number abnormalities [+4, del(6q23.3-6q25.3), +12, and +18q11-18q23] progressively increased from IgM MGUS and smoldering WM vs. symptomatic WM (18% vs. 20% and 73%, respectively; P =.008), suggesting a multistep transformation of clonal B-cells that albeit benign (i.e.: IgM MGUS and smoldering WM), already harbor the phenotypic and molecular signatures of the malignant Waldenström’s clone. Overall design: Normal bone marrow CD25+ B-cells, Clonal B-Cells from IgM Monoclonal Gammopathy of Undetermined Significance, and Clonal B-Cells from Waldenström's Macroglobulinemia...
  19. The cellular origin and malignant transformation of Waldenström's Macroglobulinemia [copy number variation] OmicsDI

    ID: E-GEOD-61650

    Date Released: 05-08-2015

    Description: clone was highly deregulated as compared to CD25-CD22+ normal B-cells, significantly less genes were differentially expressed and specific WM pathways down-regulated while comparing the transcriptome of the Waldenström’s clone vs. its normal phenotypic counterpart: CD25+CD22+dim B-cells. The frequency of specific copy number abnormalities [+4, del(6q23.3-6q25.3), +12, and +18q11-18q23] progressively increased from IgM MGUS and smoldering WM vs. symptomatic WM (18% vs. 20% and 73%, respectively; P =.008), suggesting a multistep transformation of clonal B-cells that albeit benign (i.e.: IgM MGUS and smoldering WM), already harbor the phenotypic and molecular signatures of the malignant Waldenström’s clone. Copy number analysis using the Affymetrix CytoScan 750K Array to study clonal B-cells from newly-diagnosed patients with IgM MGUS, smoldering, and symptomatic WM. Peripheral T-cells from paired and unpaired controls were also assessed....

  20. The cellular origin and malignant transformation of Waldenström's Macroglobulinemia [gene expression] OmicsDI

    ID: E-GEOD-61597

    Date Released: 05-09-2015

    Description: clone was highly deregulated as compared to CD25-CD22+ normal B-cells, significantly less genes were differentially expressed and specific WM pathways down-regulated while comparing the transcriptome of the Waldenström’s clone vs. its normal phenotypic counterpart: CD25+CD22+dim B-cells. The frequency of specific copy number abnormalities [+4, del(6q23.3-6q25.3), +12, and +18q11-18q23] progressively increased from IgM MGUS and smoldering WM vs. symptomatic WM (18% vs. 20% and 73%, respectively; P =.008), suggesting a multistep transformation of clonal B-cells that albeit benign (i.e.: IgM MGUS and smoldering WM), already harbor the phenotypic and molecular signatures of the malignant Waldenström’s clone. Normal bone marrow CD25+ B-cells, Clonal B-Cells from IgM Monoclonal Gammopathy of Undetermined Significance, and Clonal B-Cells from Waldenström's Macroglobulinemia...


Displaying 20 of 32 results for "CD22"