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Displaying 11 of 11 results for "C1QB"
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  1. C1QB_BOVIN UniProt:Swiss-Prot

    ID: Q2KIV9

    Description: Complement C1q subcomponent subunit B Collagen-like C1q Pyrrolidone carboxylic acid 4-hydroxy...

  2. C1QB_RAT UniProt:Swiss-Prot

    ID: P31721

    Description: Complement C1q subcomponent subunit B Collagen-like C1q Pyrrolidone carboxylic acid 4-hydroxy...

  3. Single Chain Recombinant Globular Head of the Complement System Protein C1q in complex with magnesium PDB

    ID: PDB:5HZF

    Description: Complement C1q subcomponent subunit A,Complement C1q subcomponent subunit ...

  4. Single Chain Recombinant Globular Head of the Complement System Protein C1q PDB

    ID: PDB:5HKJ

    Description: Complement C1q subcomponent subunit A,Complement C1q subcomponent subunit ...

  5. Data from: Analysis of the cerebrospinal fluid proteome in Alzheimer's disease Dryad

    DateIssued: 04-06-2016

    Description: er levels (p<0.05) of eight proteins (A2GL, APOM, C1QB, C1QC, C1S, FBLN3, PTPRZ, and SEZ6) in Alzheimer’s disease compared to controls using an antibody-based detection method. These proteins are involved in different biological roles spanning from cell adhesion and migration, to regulation of the synapse and the immune system....

  6. Expression data from peripheral blood OmicsDI

    ID: E-GEOD-54992

    Date Released: 07-24-2015

    Description: rculosis pleurisy from non-tuberculosis pleurisy. Complement gene expression in peripheral blood mononuclear cells of tuberculosis patients and controls were determined using whole genome transcriptional microarray assays. The mRNA and protein levels of three C1q components, C1qA, C1qB, and C1qC, were further validated by qRT-PCR and enzyme-linked immunosorbent assay, respectively. The percentages of C1q expression in CD14 positive cells were determined by flow cytometry. Finally, C1qC protein level was quantified in the pleural fluid of tuberculosis and non-tuberculosis pleurisy....

  7. Expression data from peripheral blood ArrayExpress

    ID: E-GEOD-54992

    Description: rculosis pleurisy from non-tuberculosis pleurisy. Complement gene expression in peripheral blood mononuclear cells of tuberculosis patients and controls were determined using whole genome transcriptional microarray assays. The mRNA and protein levels of three C1q components, C1qA, C1qB, and C1qC, were further validated by qRT-PCR and enzyme-linked immunosorbent assay, respectively. The percentages of C1q expression in CD14 positive cells were determined by flow cytometry. Finally, C1qC protein level was quantified in the pleural fluid of tuberculosis and non-tuberculosis pleurisy....

  8. WT vs. PPARd-/- Macrophages OmicsDI

    ID: E-GEOD-17890

    Date Released: 05-02-2014

    Description: Rdelta decreases expression of opsonins, such as C1qb, resulting in impairment of apoptotic cell clearance and reduction in anti-inflammatory cytokine production. This increases autoantibody production and predisposes global and macrophage-specific PPARdelta deficient mice to autoimmune kidney disease, a phenotype resembling the human disease systemic lupus erythematosus. Thus, PPARdelta plays a pivotal role in orchestrating the timely disposal of apoptotic cells by macrophages, ensuring that tolerance to self is maintained. HoxA9 immortalized ES cell derived myeloid precursors were created and differentiated as in Odegaard et al, 2007. After expansion, macrophages were differentiated for 8 days in the presence of MCSF in 20% L929 co...

  9. WT vs. PPARd-/- Macrophages ArrayExpress

    ID: E-GEOD-17890

    Description: Rdelta decreases expression of opsonins, such as C1qb, resulting in impairment of apoptotic cell clearance and reduction in anti-inflammatory cytokine production. This increases autoantibody production and predisposes global and macrophage-specific PPARdelta deficient mice to autoimmune kidney disease, a phenotype resembling the human disease systemic lupus erythematosus. Thus, PPARdelta plays a pivotal role in orchestrating the timely disposal of apoptotic cells by macrophages, ensuring that tolerance to self is maintained. HoxA9 immortalized ES cell derived myeloid precursors were created and differentiated as in Odegaard et al, 2007. After expansion, macrophages were differentiated for 8 days in the presence of MCSF in 20% L929 co...

  10. Expression data from peripheral blood BioProject

    ID: PRJNA238312

    Keywords: Transcriptome or Gene expression

    Access Type: download

    dataset.description: y from non-tuberculosis pleurisy. Overall design: Complement gene expression in peripheral blood mononuclear cells of tuberculosis patients and controls were determined using whole genome transcriptional microarray assays. The mRNA and protein levels of three C1q components, C1qA, C1qB, and C1qC, were further validated by qRT-PCR and enzyme-linked immunosorbent assay, respectively. The percentages of C1q expression in CD14 positive cells were determined by flow cytometry. Finally, C1qC protein level was quantified in the pleural fluid of tuberculosis and non-tuberculosis pleurisy....
  11. WT vs. PPARd-/- Macrophages BioProject

    ID: PRJNA120075

    Keywords: Transcriptome or Gene expression

    Access Type: download

    dataset.description: Rdelta decreases expression of opsonins, such as C1qb, resulting in impairment of apoptotic cell clearance and reduction in anti-inflammatory cytokine production. This increases autoantibody production and predisposes global and macrophage-specific PPARdelta deficient mice to autoimmune kidney disease, a phenotype resembling the human disease systemic lupus erythematosus. Thus, PPARdelta plays a pivotal role in orchestrating the timely disposal of apoptotic cells by macrophages, ensuring that tolerance to self is maintained. HoxA9 immortalized ES cell derived myeloid precursors were created and differentiated as in Odegaard et al, 2007. After expansion, macrophages were differentiated for 8 days in the presence of MCSF in 20% L929 co...

Displaying 11 of 11 results for "C1QB"