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Metadata

Name
To determine whether AhR KO has an effect on cell differentiation and/or global gene expression profiles in individual sorted colonic stem cells
Repository
Gene Expression Omnibus
Identifier
geo.series:GSE167595
Description
To study the role of Ahr in intestinal stem cell fate determination, we generated inducible, stem cell-specific Ahr-/- KO mice and then used scRNAseq to generate data for them. We conducted scTenifoldKnk analysis to knock out Ahr from the scGRN of WT mice and identified 53 top genes and associated pathways. We also conducted GESA analysis and identified 60 non-redundant functional gene sets, including many related to cell cycle and cell proliferation signaling pathways. It showed that enterocytes were the most significant hit, suggesting that Ahr KO induces a change in the expression of enterocyte marker genes, which may be responsible for the alteration of enterocyte differentiation fate. Taken together, our comprehensive analysis revealed the multifactorial functions of Ahr as a key player of enterocyte cell differentiation and proliferation, which is consistent with the known functions of Ahr.
Data or Study Types
Expression profiling by high throughput sequencing
Source Organization
National Center for Biotechnology Information
Access Conditions
available
Year
2021
Access Hyperlink
http://www.ncbi.nlm.nih.gov/sites/GDSbrowser?acc=GSE167595

Distributions

  • Encoding Format: Bioproject ; URL: https://www.ncbi.nlm.nih.gov/bioproject/PRJNA705096
  • Encoding Format: TXT ; URL: ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE1nnn/GSE167595/matrix/
  • Encoding Format: MINiML ; URL: ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE1nnn/GSE167595/miniml/
  • Encoding Format: SOFT ; URL: ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE1nnn/GSE167595/soft/
This project was funded in part by grant U24AI117966 from the NIH National Institute of Allergy and Infectious Diseases as part of the Big Data to Knowledge program. We thank all members of the bioCADDIE community for their valuable input on the overall project.